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1e did not reveal appropriate fits and were excluded based on incorrect fluorescence amplitudes and deviating, substrate concentration-dependent rate constants for the data sets at 1, 2.5, and 5 mM GSH

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485 These findings indicate that mitochondria iron overload, combined with ROS formation and subsequent ferroptosis, intensifies the cardiotoxic effects of DOX-induced cardiomyopathy and may be an important mechanism

Combination of in silico docking with in vivo data therefore offers a comprehensive perspective in the entirety of the molecular processes of herbal therapy

Human umbilical cord-derived mesenchymal stem cell (HUCMSC)-exosomes (EXOs) deliver miR-375 to CD4 + T cells and inhibit HDAC4 expression to increase the beclin-1 and LC3-II/LC3-I expression, promoting autophagy and inhibiting T-cell apoptosis and mitigating pathological changes, including the edema of RTECs, necrosis of renal tubules, and severe congestion induced by SAKI (Liu and Chen, 2024c)