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Figure 1 Iron metabolism At present, the exact molecular mechanism of iron overload leading to heart damage is mainly considered to be ferroptosis ( 2+ uniporter, which is essential in causing mitochondrial dysfunction and ferroptosis in cardiac iron overload ( Hepcidin, a peptide hormone originated from the liver, regulates iron absorption and distribution by suppressing the activity of transferrin ( The therapy targeting the hepcidin-transferrin axis has demonstrated initial potential, capable of effectively alleviating iron overload and ineffective erythropoiesis

Lagoda, G., Sezen, S

For infection cycles and biting, the temperature was precisely controlled at 19 C21 C and humidity at 90%-95%, maintained via automated sensor technologies under a 12-h light/dark cycle