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While smoking rates and the age at which individuals start smoking vary from country to country, our findings may be influenced by several factors such as parental, sibling, and peer smoking habits 35

A separate study found similar results suggesting an increase in MUC5AC protein levels in culture human bronchial epithelial cells exposed to PG:VG aerosols [37] as well as an increase gene expression for several inflammatory markers including cytokines interleukin-6, interleukin-8 and matrix metalloproteinase-9 (MMP-9), known to play a role in the pathogenesis of COPD [37]

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However, long-term consequences and outcomes of long-term exposure to e-cigarette aerosol are uncertain

It is the most common cause of death in CRC patients