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A long-term follow-up study showed a seven times increased risk for nonhomogenous types compared with homogenous types and a 5.4 times increased risk for lesions exceeding 200 mm 2 [38] [48]

Current dual use comprised participants who currently used both cigarettes and e-cigarettes

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Prolonged cigarette smoke exposure also switches on pro-fibrotic pathways that cause excessive extracellular matrix deposition and scar tissue formation (fibrosis) in the lungs

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