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Classifying patients according to their history of smoking has helped to understand lung cancer's gene mutations over the past decade, but researchers have found that this is not the best strategy for treating individual patients

doi:10.1016/j.marpolbul.2017.05.057

The ENaC mice, after exposure to cigarette smoke, display key pulmonary abnormalities of COPD, including inflammation, emphysema, and destruction of alveolar tissue, thus are used to study the molecular pathogenesis of COPD (25)

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