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Their mode of action may not be directly related to an AT mechanism but may involve other effects, as some of them (including PBN) were shown to release NO (nitric oxide), which can inhibit ROS-producing enzymatic activity

For example, renal endothelial cells (critical for maintaining microvascular integrity) or mesangial cells may also undergo ferroptosis in response to contrast agents, yet their role in amplifying CIN injury (e.g., via paracrine signaling or microvascular rarefaction) has not been rigorously characterized

Moreover, calcium channel blockers can mediate iron overload via divalent metal transporter-1[179], but the availability of calcium channel blockers in suppressing ferroptosis is still unknown

Oxidative stress: new insights on the association of non-alcoholic fatty liver disease and atherosclerosis

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