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Next, we investigated the mechanism by which NFT triggered the activation of Nrf2 at the molecular level

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This review highlighted that metabolic endotoxemia resulting from gut dysbiosis (e.g., LPS translocation) is a core event driving pulmonary hyperinflammation via the TLR4/NF-B pathway

These agents reduce oxidative stress, improve metabolic disturbances, mitigate lipid and glucose metabolic disorders, help alleviate pancreatic injury, inhibit inflammatory responses, and potentially prevent complications and disease progression in AP