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The salvage treatment improved the clinical, microbiological and radiological symptoms of the patients wound and overall condition

Mitochondrial Ca2dependent NLRP3 activation exacerbates the Pseudomonas aeruginosa-driven inflammatory response in cystic fibrosis

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Intracellular A in the brain possibly served as a seed mediating the formation of toxic oligomers, which then in turn were secreted to the extracellular environment and further implicated in AD pathogenesis

At doses above 10 mcg/kg, paradoxical hypertension has been observed in the same rodent preparations