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Chang et al., 2015) linked the peroxiredoxin-1 (PRX-1) status in glioma to OS caused by therapy and showed that a decreased level of PRX-1 was associated with a better response to chemotherapy (Dittmann et al., 2011) and thioredoxin (TRX) (Saitoh et al., 1998), when bound to apoptosis signal-regulating kinase 1 (ASK1), inhibited their phosphorylation and the subsequent activation of the cell death pathways that follow their dissociation under OS

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Certainly, emerging therapeutic intervention strategies for regulating ROSincluding gene therapy, stem cell therapy, and combination therapies such as antioxidant chemotherapyhave already demonstrated promising applicational potential