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It has been demonstrated that the promotion of HDAC4 phosphorylation is found after overexpression of CDKL5 via facilitating the cytoplasmic retention of HDAC4 while loss of CDKL5 leads to the decrease of HDAC4 phosphorylation, ultimately increasing the translocation of HDAC4 into the nucleus [105]

For dosing conventions used in research, see the GHK-Cu dosage reference

The analysis revealed that both the comproportionation and triad models were better suited to represent the experimental data than the reduced model, but were inconclusive with respect to overall model selection, given that both AIC/BIC values were extremely similar (Fig

This intensive approach suits short-term focused treatment phases