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Glutamine addiction extends from the context of MYC, and mutations in KRAS a key gene related to the stability and activity of c-MYC protein in cancer cells similarly cause dependence on exogenous glutamine for cellular growth and proliferation, as does MYC amplification (162, 179, 180)

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falciparum glutathione redox system comprises the electron donor NADPH, a highly active PfGR located in the cytosol and the apicoplast, and reduced/oxidized glutathione (GSH/GSSG) [21], [22], [23]

Recently endosomal processing has been demonstrated to play a pivotal role in the progression of HCC (Hu et al., 2015)