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Cellular disposition of sulphamethoxazole and its metabolites: implications for hypersensitivity

First, the molecular trigger specificity of oxalate-induced ferroptosis in renal tubular cells is undefinedhow oxalate precisely initiates the synergistic dysregulation of iron uptake (TFR1), lipid metabolism (ACSL4), and antioxidant defense (GPX4/SLC7A11) remains elusive, with no clarity on upstream signaling molecules or post-translational modifications that link oxalate exposure to ferroptosis machinery

Viral clearance was similar between wild type and heterozygous groups, but the latter had a unique type I IFN signature with an early burst of IFN followed by a decrease, different from the steady rise in wild type mice (110)