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The mechanism is plausible

There are currently no human RCTs demonstrating that systemic or intranasal administration of GHK-Cu reverses age-related cognitive decline, Alzheimers, or neurodegeneration in human subjects

Sleep deprivation increased nitrotyrosine levels in both the CA3 and DG regions of the hippocampus, and GHK treatment successfully prevented such an effect (Figure 2B)

These findings have generated significant research interest in the compound as a tool for investigating HGF/c-Met-associated signaling networks, synaptic regulatory processes, and molecular pathways governing neuronal connectivity