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In the reduced state, the thiol group of cysteinyl residue is a source of one reducing equivalent

(49) have confirmed that in vitro treatment with UA can reduce the expression levels of fibrosis markers including vimentin, discoidin domain receptor tyrosine kinase 2, tensin and Alpha-smooth muscle actin in myocardial tissues, and inhibit the transition of cardiac fibroblasts to a fibrotic state through nuclear factor erythroid 2-related factor 2 signaling pathway

Supports heart & vascular health *

This medication may interfere with certain laboratory tests (including parathyroid function), possibly causing false test results

However, a 2025 review noted only 3 RCTs, calling for larger trials (Nguyen et al., 2025)