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Specifically, mitochondrial dysfunction can lead to the production of reactive oxygen species (mtROS), which promote fibrosis, while oxidative stress imbalance can result in mitochondrial dysfunction, damage to mitochondrial DNA, and other factors that promote fibrosis [127, 128]

[DOI] [PMC free article] [PubMed] [Google Scholar] 35.Reed TT et al

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Establishing consensus on dosing parameters and administration techniques through standardized protocols is a necessary step toward reproducibility and regulatory approval (Table 5)

Importantly, overproduction of uric acid is seen in several acquired and genetic disorders with excessive rates of cell (and purine) turnover ( Table 1 - 3 )