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Collectively, these findings strongly suggest that exposure to NSW, induces systemic low-grade inflammation, likely through circadian disruption, and may contribute to the development of chronic diseases, including BC, highlighting the importance of occupational health strategies to mitigate these risks

Interestingly, the anti-apoptotic protein Bcl-2 binds to mutant SOD1 and aggregates in spinal cord mitochondria from patients and a mouse model of ALS, suggesting that mutant SOD1 may be toxic by depleting motor neurons of this anti-apoptotic protein (Pasinelli et al., 2004)

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, a hypothesis was proposed which linked chronic low dose exposure to POPs to global hypomethylation, as a consequence of GSH depletion